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Force-dependent trans-endocytosis by breast cancer cells depletes costimulatory receptor CD80 and attenuates T cell activation.

August 7, 2021

Force-dependent trans-endocytosis by breast cancer cells depletes costimulatory receptor CD80 and attenuates T cell activation.

by Park, S., Shi, Y., Kim, B. C., Jo, M. H., Cruz, L. O., Gou, Z., Ha, T., Lu, L. F., Reich, D. H. and Chen, Y.
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Reference:
Force-dependent trans-endocytosis by breast cancer cells depletes costimulatory receptor CD80 and attenuates T cell activation. (Park, S., Shi, Y., Kim, B. C., Jo, M. H., Cruz, L. O., Gou, Z., Ha, T., Lu, L. F., Reich, D. H. and Chen, Y.), In Biosens Bioelectron, 2020.
Bibtex Entry:
@Article{pmid32729511,
   Author="Park, S. and Shi, Y. and Kim, B. C. and Jo, M. H. and Cruz, L. O. and Gou, Z. and Ha, T. and Lu, L. F. and Reich, D. H. and Chen, Y.",
   Title="{Force-dependent trans-endocytosis by breast cancer cells depletes costimulatory receptor CD80 and attenuates T cell activation.}",
   Journal="Biosens Bioelectron",
   Year="2020",
   Month="Oct",
   URL="https://www.ncbi.nlm.nih.gov/pubmed/32729511",
}

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